Instantaneous current efficiency decreased with increase

in current density.

CONCLUSIONS: The degradation of pollutants occurred mainly at the boundary layer between the electrode and the bulk solution. The indirect oxidation by active chlorine generated from the chlorides was proven to be the primary mechanism of electrochemical oxidation treatment. The removal of COD(Cr) in this study followed a pseudo-first-order kinetic model. (C) 2011 Society of Chemical Industry”
“Microbial biotransformation of acetyl-11-keto-boswellic acid by Cunninghamella elegans AS 3.1207 was carried out, and totally four transformed products were isolated. On the basis of the extensive spectral data, their structures were characterized as 7-hydroxy-11-keto-boswellic acid (1), 7,30-dihydroxy-11-keto-boswellic acid (2), 7,16-dihydroxy-3-acetyl-11-keto-boswellic Z-IETD-FMK datasheet acid (3), and 7,15,21-trihydroxy-3-acetyl-11-keto-boswellic acid (4), respectively. Among them, products 1 and 2 are the new compounds.”
“Case Description-A

4-year-old 29-kg (63.8-Ib) spayed female Husky crossbred was referred for emergency treatment because of catastrophic hemorrhagic shock following Compound C in vivo attempts at cystocentesis for investigation of suspected urinary tract infection.

Clinical Findings-On arrival at the hospital, clinicopathologic assessments revealed rapidly decreasing PCV and worsening hypoproteinemia, compared with findings immediately prior to referral. The dog had severe hyperlactemia. Ultrasonography revealed the presence of free fluid in the abdomen; the fluid appeared to be blood (determined via abdominocentesis).

Treatment and Outcome-Urgent surgical exploration was undertaken. Two small lacerations in the ventral aspect of the abdominal aorta just dorsal to the bladder were identified and repaired. Multiple transfusions of packed RBCs (5 units) and fresh frozen plasma (3 units) were administered, and autotransfusion of blood (1.2 L) from the abdomen was performed. The dog recovered well from surgery and anesthesia, selleck compound but

developed signs of severe pain and swelling of both hind limbs, which were attributed to reperfusion injury following aortic occlusion during surgery. Treatment included administration of S-adenosylmethionine (23 mg/kg [10.5 mg/lb], PO, q 24 h) and analgesia; 5 days after surgery, the hind limb problems had resolved and treatments were discontinued.

Clinical Relevance-In the clog of this report, aortic laceration secondary to cystocentesis was successfully treated with a combination of surgery and massive transfusion; the development of reperfusion injury was an interesting and reversible complication of surgery. The possibility of damage to intra-abdominal structures should be investigated if a dog becomes acutely ill after cystocentesis.